1. Ivermectin prevents the virus from entering target body cells even if it has already entered through the mouth, nose, or eyes.

It binds into the spike - the corona - protein, making it unable to key into ACE-2 receptors at the surface of the cells and making it open (https://pubmed.ncbi.nlm.nih.gov/32871846/). It’s like a key smeared with epoxy that has hardened, it cannot fit into the keyhole anymore. This includes emerging strains with mutations on the spike protein that threaten to disable existing vaccines.

Unlike the vaccines, this allows the immune system to develop antibodies – immunity - for each emerging variant spike protein encountered

(https://www.who.int/news-room/feature-stories/detail/how-do-vaccines-work).

2 - Even with the virus already inside the cells, Ivermectin prevents further entry to the cell nucleus to control it and shut down the immune response. This is why initially, patients don’t feel anything.

Ivermectin binds into Importin α/β1 heterodimer protein which the virus uses as the transport to enter the cell's control center - its nucleus (https://pubmed.ncbi.nlm.nih.gov/22417684/).

3 - Ivermectin prevents the virus from replicating by inhibiting the Helicase enzyme it needs to do so. This is why Ivermectin may also be effective against some forms of cancer.

Ivermectin inhibits the Helicase enzyme necessary to initiate genomic transcription and eventually complete replication through protein translation (https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5835698/)

4 - Ivermectin prevents COVID-19-induced Cytokine Storm that triggers respiratory distress (ARDS) and attacks of immune cells of healthy organs resulting in organ damage, failure, and mortality.

A cytokine storm is the release of a large amount of pro-inflammatory signaling molecules called cytokines, causing multisystem organ failure and death. In SARS-COV-2, it also causes the leakage of fluids into the alveolar spaces in the lungs, blocking Oxygen intake. Ivermectin is an immuno-modulator that suppresses cytokine storms (https://pubmed.ncbi.nlm.nih.gov/19109745/)

5 - A fatal effect of SARS-COV-2 infection is the blood becoming sticky and blocking blood vessels and Oxygen from being delivered to cells.

SARS-COV-2 infection uses the protein receptor CD147 in the membranes of red blood cells to make the blood sticky and cause blockage in blood vessels. Ivermectin alleviates CD147-mediated blood vessel blockage and blood clots (https://papers.ssrn.com/sol3/papers.cfm?abstract_id=3636557).

6 - Ivermectin increases Interferon production and enhances its effects.

Interferons are like bullets fired by the body's immune system that destroy viruses like SARS-COV-2 upon entry. But when the viruses are so many that they overwhelm the body's immune system, the undestroyed viruses stick to the target cells and get in to use them to multiply and infect other cells. Increased Interferon production and enhancement in its effects were observed in Onchocerciasis patients administered with Ivermectin (https://pubmed.ncbi.nlm.nih.gov/7930742/)

Ivermectin prevents infection. It shortens the period of viral shedding making one stop transmitting the virus sooner. It is an effective anti-viral and an anti inflammatory drug. Negating Ivermectin's capacities and not using it to save others is inhuman